I appreciate that your cracking seems to relate to sa sense of inflammation but it is hard to see how this works. Inflammation makes things swollen and stiff. It reduces range of mobility and the likelihood of bones shifting out of place. You may get inflammation as a reaction to a subluxation but inflammation does not encourage subluxation. It is a bit like the difference between a plastic drink bottle when full of lemonade and one that you have squeezed empty and screwed the lid on. The full one will not crack but the empty one will if you press it - and go into kinked shapes. People with gross inflammation from rheumatoid arthritis do not get cracking in their joints - just grinding if the cartilage surface has been eaten away and worn down.
Well, I've learned to not refer to inflammation as if it is a single thing. (E.g., MCs have many differet products with different effects.) I'm referring to mast cell proteases presumably weakening the supporting tendons, thereby allowing the joint to more readily go out of place. Not to hostamine and downstream signallers creating increased permeability. Btw, I only have had bad joint problems after 4 years of getting CFS, so it's not even been consistent in me.
I've also had plenty of experience with tendons getting weaker during inflammation - in that I could lift less weight.
"It is a bit like the difference between a plastic drink bottle when full of lemonade and one that you have squeezed empty and screwed the lid on. The full one will not crack but the empty one will if you press it - and go into kinked shapes."
I'm generally referring to use of force large enough to overcome any hydraulic type of immobilization.
"People with gross inflammation from rheumatoid arthritis do not get cracking in their joints - just grinding if the cartilage surface has been eaten away and worn down."
Yep, but I'm not referring to the immune system destroying cartilage; I'm referring to temporary weakening of tendons.