Firestormm
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Only read the first page - seems right up my street - will read the rest later. Thought others might be interested.
Sleep Apnea is a common enough missed diagnosis in patients with ME as it is others I think - though in a recent study from Newton she found it was one of the most common misdiagnoses. I have recently been diagnosed with chronic obstructive sleep apnea myself - after 10 years of asking for a sleep study. Am awaiting treatment to commence.
Of course the difficulty facing those of us with ME is that sleep disruption - too much, too little - and 'unrefeshing sleep' has been seen as part and parcel of the disease. There may be a cross-over with the reported effects of sleep apnea, or it may be that more of us meet the criteria (and testing) for apnea than we realise.
Treatment can involve I think two main avenues: one is a 'mouth guard' to open the airways at night, and the other is nighttime oxygen delivered through a mask (which is the treatment I am lined up for I believe) which attempts to force a constant supply of oxygen down the throat.
Sleep apnea is essentially oxygen starvation to the brain resulting in the brain telling the body to breathe, because the body - for several reasons - stops breathing for short periods within (for me) more significant periods during the night.
So it's often a case of 'snoring' to take a deep breath, and the lack of oxygen and resulting impulse to suddenly 'wake' can result in 'episodes' or interruptions to the extent that it is thought (in my case), I am experiencing seizures.
But they aren't really sure. It does 'interrupt' your sleep rhythms leading to feelings of 'fatigue' during the day, and as can be seen from the paper - cognitive difficulties and (it is speculated) degeneration.
The symptoms are obviously of interest to those with ME and to those working on research into the condition.
I need to read more about this one though:
Sleep Apnea is a common enough missed diagnosis in patients with ME as it is others I think - though in a recent study from Newton she found it was one of the most common misdiagnoses. I have recently been diagnosed with chronic obstructive sleep apnea myself - after 10 years of asking for a sleep study. Am awaiting treatment to commence.
Of course the difficulty facing those of us with ME is that sleep disruption - too much, too little - and 'unrefeshing sleep' has been seen as part and parcel of the disease. There may be a cross-over with the reported effects of sleep apnea, or it may be that more of us meet the criteria (and testing) for apnea than we realise.
Treatment can involve I think two main avenues: one is a 'mouth guard' to open the airways at night, and the other is nighttime oxygen delivered through a mask (which is the treatment I am lined up for I believe) which attempts to force a constant supply of oxygen down the throat.
Sleep apnea is essentially oxygen starvation to the brain resulting in the brain telling the body to breathe, because the body - for several reasons - stops breathing for short periods within (for me) more significant periods during the night.
So it's often a case of 'snoring' to take a deep breath, and the lack of oxygen and resulting impulse to suddenly 'wake' can result in 'episodes' or interruptions to the extent that it is thought (in my case), I am experiencing seizures.
But they aren't really sure. It does 'interrupt' your sleep rhythms leading to feelings of 'fatigue' during the day, and as can be seen from the paper - cognitive difficulties and (it is speculated) degeneration.
The symptoms are obviously of interest to those with ME and to those working on research into the condition.
I need to read more about this one though:
Daytime Impairment and Neurodegeneration in OSAS
December 2013
Magdalena Nowak, MD; Johannes Kornhuber, MD; Robert Meyrer, MD
Department of Psychiatry, University of Erlangen, Germany
Study Objective:
Controversy surrounds the pathogenesis of neurocognitive daytime dysfunction exhibited by patients with o bstructive sleep apnea syndrome (OSAS).
Underlying brain dysfunctions and damage have long been suspected as a cause of some of this impairment. Neuroimaging has enabled scientists to test these long-held theories.
This paper is based on a comprehensive review of recent publications on neuroimaging studies in this area. It seeks to highlight results of recent research, which suggest connections between persistent neurocognitive daytime impairment of executive functions, underlying signs of cerebral metabolic impairment and neurodegeneration, considering possible cerebrovascular impairment in OSAS patients.
We propose the existence of a neurodegenerative process.
Full Paper